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Tryptophan as a Link between Psychopathology and Somatic States

Sascha Russo, MD, Ido P. Kema, PhD, M. Rebecca Fokkema, MSc, Jim C. Boon, MD, Pax H. B. Willemse, MD, PhD, Elisabeth G. E. de Vries, MD, PhD, Johannes A. den Boer, MD, PhD and Jakob Korf, PhD

From Department of Biological Psychiatry (S.R., J.C.B., J.A.D.B., J.K.), Department of Laboratory Medicine (I.P.K., M.R.F.), Department of Medical Oncology (P.H.B.W., E.G.E.D.V.), University Hospital Groningen, The Netherlands



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  		Fig. 1. Figure summarized model of oxidative (down arrow) and hydroxylase (right arrow) pathways of TRP. * = Tetrahydrobiopterin-dependent reaction; + = pyridoxal phosphate dependent reaction.

 


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  		Fig. 2. Figure metabolism of tryptophan under normal, inflammatory, and hormonally induced situations. Size of arrows indicates quantitative significance.

 




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